Nuki G., Simkin P. A. A Concise History of Gout and Hyperuricemia and Their Treatment // Arthritis Research & Therapy. 2006. Vol. 8. Suppl. 1. P. S1 // doi.org/10.1186/ar1906 (http://doi.org/10.1186/ar1906).
9
Maurer J. Early Gout Is Bad for the Heart: Recent Research Context // Med-Page Today, November 28, 2019 // medpagetoday.com/reading-room/acrr/generalrheumatology/83581 (http://medpagetoday.com/reading-room/acrr/generalrheumatology/83581). См. также Li Y. et al. Clinical Characteristics of Early-Onset Gout in Outpatient Setting // ACR Open Rheumatology. 2019. Vol. 1. № 7. Pp. 397–402 // doi.org/10.1002/acr2.11057 (http://doi.org/10.1002/acr2.11057).
10
Singh J. A. Gout: Will the “King of Diseases” Be the First Rheumatic Disease to Be Cured? // BMC Medicine. 2016. Vol. 14. P. 180 // doi.org/10.1186/s12916-016-0732-1 (http://doi.org/10.1186/s12916-016-0732-1).
11
George Ch., Minter D. A. Hyperuricemia // StatPearls. Treasure Island, FL: 2021 // ncbi.nlm.nih.gov/books/NBK459218/ (http://ncbi.nlm.nih.gov/books/NBK459218/).
12
Chen J.-H. et al. Serum Uric Acid Level as an Independent Risk Factor for All-Cause, Cardiovascular, and Ischemic Stroke Mortality: A Chinese Cohort Study // Arthritis & Rheumatology. 2009. February. Vol. 61. № 2. Pp. 225–232 // doi.org/10.1002/art.24164 (http://doi.org/10.1002/art.24164). См. также Prado de Oliveira E., Burini R. C. High Plasma Uric Acid Concentration: Causes and Consequences // Diabetology & Metabolic Syndrome. 2012. April. Vol. 4. P. 12 // doi.org/10.1186/1758-5996-4-12 (http://doi.org/10.1186/1758-5996-4-12).
13
El Ridi R., Tallima H. Physiological Functions and Pathogenic Potential of Uric Acid: A Review // Journal of Advanced Research. 2017. September. Vol. 8. № 5. Pp. 487–493 // doi.org/10.1016/j.jare.2017.03.003 (http://doi.org/10.1016/j.jare.2017.03.003).
14
El Ridi and Tallima, Physiological Functions and Pathogenic Potential of Uric Acid.
15
DiNicolantonio J. J., O’Keefe J. H., Lucan S. C. Added Fructose: A Principal Driver of Type 2 Diabetes Mellitus and Its Consequences // Mayo Clinic Proceedings. 2015. March. Vol. 90. № 3. Pp. 372–381 // doi.org/10.1016/j.mayocp.2014.12.019 (http://doi.org/10.1016/j.mayocp.2014.12.019).
16
Stirpe F. et al. Fructose-induced Hyperuricaemia // The Lancet. 1970. December. Vol. 296. № 7686. Pp. 1310–1311 // doi.org/10.1016/s0140-6736(70)92269-5 (http://doi.org/10.1016/s0140-6736%2870%2992269-5).
17
Goran M. I. et al. The Obesogenic Effect of High Fructose Exposure During Early Development // Nature Reviews Endocrinology. 2013. August. Vol. 9. № 8. Pp. 494–500.
18
Rivard Ch. et al. Sack and Sugar, and the Aetiology of Gout in England Between 1650 and 1900 // Rheumatology. 2013. March. Vol. 52. № 3. Pp. 421–426 // doi.org/10.1093/rheumatology/kes297 (http://doi.org/10.1093/rheumatology/kes297).
19
Zgaga L. et al. The Association of Dietary Intake of Purine-Rich Vegetables, Sugar-Sweetened Beverages and Dairy with Plasma Urate, in a Cross-Sectional Study // PLOS ONE. 2012. Vol. 7. № 6. P. e38123 // doi.org/10.1371/journal.pone.0038123 (http://doi.org/10.1371/journal.pone.0038123).
20
Singh J. A., Reddy S. G., Kundukulam J. Risk Factors for Gout and Prevention: A Systematic Review of the Literature // Current Opinion in Rheumatology. 2011. March. Vol. 23. № 2. Pp. 192–202 // doi.org/10.1097/BOR.0b013e3283438e13 (http://doi.org/10.1097/BOR.0b013e3283438e13).
21
LUV созвучно английскому слову love (любовь). Прим. пер.
22
Выражение автора, означающее, что мочевая кислота разрушает организм, как бомба. Прим. пер.
23
Enzinger Ch. et al. Risk Factors for Progression of Brain Atrophy in Aging: Six-Year Follow-Up of Normal Subjects // Neurology. 2005. May 24. Vol. 64. № 10. Pp. 1704–1711 // doi.org/10.1212/01.WNL.0000161871.83614.BB (http://doi.org/10.1212/01.WNL.0000161871.83614.BB).
24
Crane P. K. et al. Glucose Levels and Risk of Dementia // New England Journal of Medicine. 2013. August. Vol. 369. № 6. Pp. 540–548 // doi.org/10.1056/NEJMoa1215740 (http://doi.org/10.1056/NEJMoa1215740).
25
Van Pelt G. W. A Study of Haig’s Uric Acid Theory // Boston Medical and Surgical Journal. 1896. Vol. 134. № 6. Pp. 129–134 // doi.org/10.1056/NEJM189602061340601 (http://doi.org/10.1056/NEJM189602061340601).
26
Johnson R. J. et al. Lessons from Comparative Physiology: Could Uric Acid Represent a Physiologic Alarm Signal Gone Awry in Western Society? // Journal of Comparative Physiology B: Biochemical, Systemic, and Environmental Physiology. 2009. January. Vol. 179. № 1. Pp. 67–76 // doi.org/10.1007/s00360-008-0291-7 (http://doi.org/10.1007/s00360-008-0291-7).
27
Framingham Heart Study // framinghamheartstudy.org (http://framinghamheartstudy.org/).
28
Culleton B. F. et al. Serum Uric Acid and Risk for Cardiovascular Disease and Death: The Framingham Heart Study // Annals of Internal Medicine. 1999. July. Vol. 131. № 1. Pp. 7–13 // doi.org/10.7326/0003-4819-131-1-199907060-00003 (http://doi.org/10.7326/0003-4819-131-1-199907060-00003).
29
Неполный список исследовательских работ доктора Ричарда Джонсона есть на его странице scholar.google.com/citations?user=dTgECeMAAAAJ&hl=en (http://scholar.google.com/citations?user%3ddTgECeMAAAAJ%26hl%3den).
30
Johnson R. J., Andrews P. The Fat Gene // Scientific American. 2015. October. Vol. 313. № 4. Pp. 64–69 // doi.org/10.1038/scientificamerican1015-64 (http://doi.org/10.1038/scientificamerican1015-64).
31
Johnson and Andrews. The Fat Gene.
32
Johnson and Andrews. The Fat Gene.
33
